To understand leaky gut, you need to understand the gut’s delicate inner lining. This special barrier is strong, but ultra-thin. In fact, it is only one cell layer thick. “Good” digestive bacteria ...

"Leaky gut” is a physiological process, not a formal medical diagnosis. Q: I keep hearing about “leaky gut” and the symptoms it causes. What is it exactly? What’s the best way to fix it? A: “Leaky gut ...

Leaky gut occurs when your gut barrier becomes compromised, allowing substances to “leak” through. Diarrhea, bloating, brain fog or widespread pain or inflammation are common symptoms of leaky gut.

If you've ever been on #GutTok, the popular corner of TikTok dedicated to gut health, chances are you've heard people talk about "leaky gut" or "leaky gut syndrome." But what exactly is it? Leaky gut ...

Leaky gut syndrome describes a weakness in your intestinal barrier that may allow toxins to enter your bloodstream. This may cause hives and other skin problems in some people. Dietary changes may ...

Gut health: you’re either someone who’s considering what probiotic to buy next, or, you’ve got zero clue about the state of play in one of your body’s most important microbiomes. Our gut health ...

Leaky gut syndrome, or increased intestinal permeability, is a condition where the lining of the small intestine becomes damaged and overly porous, allowing undigested food particles, toxins and ...

The amount of time it takes to heal from a “leaky gut” varies based on a person’s symptoms, overall health, and the underlying cause. “Leaky gut” is not a recognized medical diagnosis, and it is ...

As interest in living longer – and staying healthier into old age – continues to grow, doctors are increasingly looking beyond obvious lifestyle factors such as diet and exercise. One area now ...

A study published in eGastroenterology investigated the role of cannabinoid receptor 1 (CBR1) in intestinal permeability, commonly called “leaky gut.” In particular, the study focused on CBR1’s ...

Alcohol binge increases intestinal permeability by activating intestinal epithelial CB1R-ERK1/2 signalling with subsequently reduced differentiation and downregulation of tight junctions (left).