Left Ventricular Thrombus Post-MI: A Critical Complication After an acute MI, the injured myocardium can create a pro-thrombotic environment, leading to left ventricular thrombus (LVT) formation. Key Points: · Risk Period: Highest in the first weeks following a large anterior ST-elevation MI. · Primary Concern: Systemic embolization, most critically causing stroke. · Management: Requires prompt imaging (echo/CMR) and immediate initiation of anticoagulation therapy. Awareness, early detection, an

2.9K views · 24 reactions | Ultrasound Corner: Echocardiographic Image of Left Ventricular Thrombus Formation During Cardiac Contractility Recovery Under Extracorporeal Membrane Oxygenation Support What's the diagnosis? Read the full research in the February issue: https://hubs.la/Q037rfwg0 #MedEd #JournalCHEST | American College of Chest Physicians | Facebook

Anteroapical thrombus in patient with anteroapical akinesia post anteroapical MI Why an akinetic area predisposes to thrombus formation 1. Blood stasis (the main reason) An akinetic region does not contract, so it fails to eject blood properly. Blood becomes stagnant in that pocket, especially in the apex → slow-flow / spontaneous echo contrast appears on echo. Slow flow is the perfect environment for clot formation. 2. Endocardial injury after infarction Akinetic segments usually represent an a

Left ventricular failure (LVF) is characterized by symptoms resulting from the inability of the left ventricle to pump blood efficiently, leading to congestion in the lungs. Classical signs include shortness of breath, orthopnea (difficulty breathing when lying flat), paroxysmal nocturnal dyspnea (sudden nighttime breathlessness), and fatigue. On physical examination, the patient may exhibit crackles (crepitations) on auscultation of the lungs, indicating pulmonary congestion. Swelling in the le